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Sildenafil is often a potent and selective inhibitor of cGMP-specific phosphodiesterase type 5 (PDE5), which can be liable for degradation of cGMP in the corpus cavernosum. The molecular structure of sildenafil is comparable to that of cGMP and acts being a aggressive binding agent of PDE5 during the corpus cavernosum, leading to much more cGMP and

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